A team of American researchers has concluded that chronic high blood sugar can impair memory and lead to Alzheimer’s disease.
A new study has found that type 2 diabetes may increase the risk of Alzheimer’s disease by altering some brain functions, according to the US website UPI.
And the site indicated, in a report published on Saturday, that a team of researchers at the University of Nevada in the United States found that chronic rise in blood sugar levels can impair memory and change the brain functions responsible for it.
The website quoted study author Dr. James Hyman as saying: “Diabetes is a major risk factor for Alzheimer’s disease, but the reason behind this was not clear. Similar to what is observed in Alzheimer’s disease models.
“This study is the first to show evidence of alterations in neural activity due to the similarity between outcomes caused by elevated blood glucose levels with those observed in models of Alzheimer’s disease,” Heymann added.
In the study, which was conducted on mice, the research team found that two parts of the brain essential for forming and retrieving memories, the hippocampus and the anterior cingulate cortex, are over-connected with type 2 diabetes.
The researchers say that when these two areas of the brain, which are usually affected early in Alzheimer’s disease, need to access the correct information and complete a task, they over-communicate with each other, causing errors.
“We know that having synchronization is important for different parts of the brain to work together, but we’re finding more synchronization these days, but the most important thing about that synchronization is that it has to happen at the right time,” Hayman explains. Sometimes, there are too many connections between certain areas of the brain which we think leads to memory difficulties, among other things.”
Hayman believed that Alzheimer’s patients may have excessive connectivity in some areas of the brain that require some flexibility, explaining that the study he conducted with his colleagues, whose results were published in the journal Communications Biology, also found evidence of this.